I've been casually daydreaming about possible paths an "AI augmented" newsfeed experience could take.
I'm imagining an initial group of agents scraping the web for events (twitter/x, science/eng/business publications) that could potentially translate to stories that their end users (humans) might be interested in.
those stories get picked up by another set of bots (equivalent to reddit posters) and get published on an agent-only social network where a diverse set of agents with different "backgrounds"/personalities comment and discuss on the story.
inputs from this post (article + comments) are picked up by an 'editor' agent and go into a final summarized article designed for human eyes or humans personal agent or newsfeed agent.
humans being the end users browse their (ai enhanced) newsfeed which has its own private, continuously evolving algorithms based on knowledge of 1) what and 2) how its human likes to consume/think.
information is "backpropagated" to inform/reinforce the initial scraper group of bots on what to look out for.
Some humans produce slop sometimes, but AI produces slop basically always. I don't see that as an argument to just add more into the system because humans do sometimes.
Your application also doesn't "serve human values". It just statistically gives someone content it thinks they'll want to hear/read, and hides content that might make them uncomfortable or question their own views. A self-reinforcing echo chamber, like I said.
I know AI is all the rage and everyone is trying to come up with something actually useful for it, but this isn't it.
The article describes the mechanism in some detail near the end. As I understand it, it's not really "coordination" in the sense that they exchange messages through the electricity.
It's more that every cell has to maintain a voltage difference between the inside and outside ("membrane potential"). A healthy cell does that constantly using "ion pumps" that use chemical energy (ATP) to increase the potential.
If that potential falls below a certain threshold, certain molecular mechanisms (voltage-sensitive ion channels) inside the cell are triggered that lead to ejection.
Interestingly, are also other mechanisms (pressure-sensitive ion channels) that will "intentionally" make it harder for a cell to maintain its potential if it's already weakened or if the surrounding region is very crowded.
As such, I think the effect of current would depend on the way how it would change the voltage differences of the individual cells.
I wonder what the role of inflammation in all this is. It must be a major disrupter (or the effect?) of such electrical comms, with all these cytokines and fluid influx changing things around.